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BackgroundIgG4-related disease is a multiorgan, relapsing, fibroinflammatory, immune-mediated disorder with no approved therapy. Inebilizumab targets and depletes CD19+ B cells and may be effective for treating patients with IgG4-related disease.MethodsIn this phase 3, multicenter, double-blind, randomized, placebo-controlled trial, adults with active IgG4-related disease underwent randomization in a 1:1 ratio to receive inebilizumab (300-mg intravenous infusions on days 1 and 15 and week 26) or placebo for a 52-week treatment period. Participants in both groups received identical glucocorticoid tapers. Glucocorticoids were allowed to treat disease flares, but background immunosuppressants were not permitted. The primary end point was the first treated, adjudicated disease flare during the treatment period, assessed in a time-to-event analysis. Key secondary end points were the annualized flare rate and treatment-free and glucocorticoid-free complete remission.ResultsA total of 135 participants with IgG4-related disease underwent randomization: 68 participants were assigned to receive inebilizumab and 67 were assigned to receive placebo. Treatment with inebilizumab reduced flare risk; 7 participants (10%) in the inebilizumab group had at least one flare, as compared with 40 participants (60%) in the placebo group (hazard ratio, 0.13; 95% confidence interval [CI], 0.06 to 0.28; P<0.001). The annualized flare rate was lower with inebilizumab than with placebo (rate ratio, 0.14; 95% CI, 0.06 to 0.31; P<0.001). More participants in the inebilizumab group than in the placebo group had flare-free, treatment-free complete remission (odds ratio, 4.68; 95% CI, 2.21 to 9.91; P<0.001) and flare-free, glucocorticoid-free complete remission (odds ratio, 4.96; 95% CI, 2.34 to 10.52; P<0.001). Serious adverse events occurred during the treatment period in 12 of the participants (18%) who received inebilizumab and 6 of the participants (9%) who received placebo.ConclusionsInebilizumab reduced the risk of flares of IgG4-related disease and increased the likelihood of flare-free complete remission at 1 year, confirming the role of CD19-targeted B-cell depletion as a potential treatment for IgG4-related disease. (Funded by Amgen; MITIGATE ClinicalTrials.gov number, NCT04540497.).

Original publication

DOI

10.1056/nejmoa2409712

Type

Journal article

Journal

The New England journal of medicine

Publication Date

11/2024

Addresses

From the Division of Rheumatology, Allergy, and Immunology, Massachusetts General Hospital, Harvard Medical School, Boston (J.H.S., Z.S.W., C.A.P.); the Division of Rheumatology, Emory University School of Medicine, Atlanta (A.K.); the Department of Rheumatology, Peking Union Medical College Hospital, Chinese Academy of Medical Science, National Clinical Research Center for Dermatologic and Immunologic Diseases, Beijing (W.Z.), and the Department of Rheumatology and Immunology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan (L.D.) - both in China; the Unit of Immunology, Rheumatology, Allergy, and Rare Diseases, IRCCS San Raffaele Scientific Institute, Milan (E.D.T., M.L.); the Department of Internal Medicine, Kansai Medical University Kori Hospital, Osaka (K.O.), the First Department of Internal Medicine, University of Occupational and Environmental Health, Kitakyushu (Y.T.), and Nagahama City Hospital, Nagahama (H.U.) - all in Japan; the Department of Clinical Science, Intervention, and Technology, Karolinska Institutet, Stockholm (J.M.L.); Aix-Marseille Université, Assistance Publique-Hôpitaux de Marseille, Centre National de la Recherche Scientifique, INSERM, Centre d'Immunologie de Marseille-Luminy, Hopital de la Timone, Internal Medicine Department, Marseille (N.S., M.E.), and the Pancreatology and Digestive Oncology Department, Beaujon Hospital, Université Paris Cité, Clichy (V.R.) - both in France; the Department of Gastroenterology, University College London Hospitals, London (G.J.W.), the Hepato-Pancreato-Biliary Unit, Freeman Hospital, Newcastle upon Tyne (M.K.N.), and the Translational Gastroenterology and Liver Unit, John Radcliffe Hospital, and Nuffield Department of Medicine, University of Oxford, Oxford (E.L.C.) - all in the United Kingdom; the Internal Medicine Department, Vall d'Hebron Hospital, Vall d'Hebron Research Institute, Universitat Autònoma de Barcelona, Barcelona (F.M.V.); and Amgen, Thousand Oaks, CA (X.D., Y.W., Q.L., N.R., D.C.).

Keywords

MITIGATE Trial Investigators